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Oxidation Δm +16 severity: high

Trp oxidation (kynurenine, oxindolylalanine, NFK)

The indole of Trp is highly susceptible to oxidative attack — gives oxindolylalanine (+16), kynurenine (+4 after ring rearrangement), or N-formylkynurenine (+32). Often appears as a peak cluster at +16/+4/+32.

Affected residue(s): W
Other mass signatures from the same mechanism:
  • +4 — +4 Da, Trp → Kynurenine (after rearrangement)
  • +31.9898 — +32 Da, N-formylkynurenine

Why it happens (mechanism)

The C2-C3 indole double bond is electron-rich; singlet O₂ or peroxide attacks it. Initial product is 2- or 3-oxindolylalanine (+16 Da). Further oxidative cleavage of the ring + rearrangement gives kynurenine (the indole N→formamide, then loss of CO gives +4 net) or N-formylkynurenine (+32).

When it strikes (triggers)

TFA cleavage *without sufficient scavenger*. Long cleavage time. Light. Air. H₂O₂. Photo-oxidation during HPLC under UV.

How to spot it (MS signature)

+15.99 (oxindolylalanine), +3.99 (Kyn), +31.99 (NFK). The whole cluster is a fingerprint of Trp oxidation. MS/MS shows characteristic 2-aminobenzoyl fragment for Kyn.

How to prevent it

If it already happened (salvage)

Source

Yi Yang, Side Reactions in Peptide Synthesis (Elsevier, 2016), Chapter 9, §9.3.